Defective Osteogenic Differentiation in the Development of Osteosarcoma

نویسندگان

  • Eric R. Wagner
  • Gaurav Luther
  • Gaohui Zhu
  • Qing Luo
  • Qiong Shi
  • Stephanie H. Kim
  • Jian-Li Gao
  • Enyi Huang
  • Yanhong Gao
  • Ke Yang
  • Linyuan Wang
  • Chad Teven
  • Xiaoji Luo
  • Xing Liu
  • Mi Li
  • Ning Hu
  • Yuxi Su
  • Yang Bi
  • Bai-Cheng He
  • Ni Tang
  • Jinyong Luo
  • Liang Chen
  • Guowei Zuo
  • Richard Rames
  • Rex C. Haydon
  • Hue H. Luu
  • Tong-Chuan He
چکیده

Osteosarcoma (OS) is associated with poor prognosis due to its high incidence of metastasis and chemoresistance. It often arises in areas of rapid bone growth in long bones during the adolescent growth spurt. Although certain genetic conditions and alterations increase the risk of developing OS, the molecular pathogenesis is poorly understood. Recently, defects in differentiation have been linked to cancers, as they are associated with high cell proliferation. Treatments overcoming these defects enable terminal differentiation and subsequent tumor inhibition. OS development may be associated with defects in osteogenic differentiation. While early regulators of osteogenesis are unable to bypass these defects, late osteogenic regulators, including Runx2 and Osterix, are able to overcome some of the defects and inhibit tumor propagation through promoting osteogenic differentiation. Further understanding of the relationship between defects in osteogenic differentiation and tumor development holds tremendous potential in treating OS.

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عنوان ژورنال:

دوره 2011  شماره 

صفحات  -

تاریخ انتشار 2011